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Fenofibrate and fenofibric acid are used as adjuncts to dietary therapy in the management of primary hypercholesterolemia and mixed dyslipidemia. The drugs also are used as adjuncts to dietary therapy in the management of severe hypertriglyceridemia. Drug therapy is not a substitute for, but an adjunct to, these nondrug therapies and measures, which should be continued when drug therapy is initiated. The effects of fenofibrate and fenofibric acid on cardiovascular morbidity and mortality or noncardiovascular mortality have not been established. Fenofibrate and fenofibric acid are used as adjuncts to dietary therapy to decrease elevated serum total and low-density lipoprotein LDL -cholesterol, triglyceride, and apolipoprotein B apo B concentrations, and to increase high-density-lipoprotein HDL -cholesterol concentrations in the management of primary hypercholesterolemia and mixed dyslipidemia, including heterozygous familial hypercholesterolemia and other causes of hypercholesterolemia. Efficacy and safety of fenofibrate and fenofibric acid in the management of hypercholesterolemia were established in 4 randomized, double-blind, placebo-controlled studies of months' duration in patients with primary hypercholesterolemia or mixed dyslipidemia. Efficacy and safety of fenofibric acid in combination with a statin were established in 3 randomized, double-blind studies of 12 weeks' duration and a long-term week open-label extension study in patients with mixed dyslipidemia.

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Your medication may look different. If you have questions, ask your pharmacist. Generic name: Fenofibrate - oral. Pronunciation fen-oh-FYE-brate. It works by increasing the natural substance enzyme that breaks down fats in the blood. Fenofibrate belongs to a group of drugs known as fibrates.

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A literature search was conducted, focusing on comparative studies examining bioavailability, food effects, absorption, and lipid efficacy. Fenofibrate is highly lipophilic, virtually insoluble in water, and poorly absorbed. Furthermore, a more recent observational study in an academic cardiology clinic found that Factors contributing to residual risk include poor nutrition, lack of exercise, and failure to adequately normalize lipoproteins other than LDL-C. While 2 of these residual risk factors are modifiable by lifestyle changes, an alternative therapeutic intervention strategy is sometimes necessary to correct mixed lipid abnormalities.

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