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Soma carisoprodol is a pain reliever of relaxing nature originally approved by the FDA in Currently, it is manufactured by Meda Pharmaceuticals and remains high on the list of highly potent yet commonly abused medications. Spasticity that has UMNS as its underlying cause which is a set of certain indications connected with disordered reflexes, bad dexterity, repetitive or twisting movements, overreacting to external or internal stimuli, impaired or weak movements, contractures, and rapid onset of tiredness often starts from a variety of disorders affecting the spinal cord or cortex. Among those spasticity-associated disorders there are multiple sclerosis MS, brain damage, spinal cord damage, post-stroke condition, and cerebral palsy.
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Introduction: Carisoprodol is a centrally acting muscle relaxant used in the treatment of various musculoskeletal disorders whose main metabolite, meprobamate, is a controlled substance in the United States due to its sedative properties and potential for abuse. At hospital discharge, his cognitive function significantly improved compared with when he was on carisoprodol. Conclusion: Carisoprodol withdrawal is an important and under-recognized syndrome that should be considered in patients presenting with neurologic symptoms who are taking the medication.
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Mood disorders are associated with changes in regional brain function, which is different to those seen in non-depressed patients who feel sad. Newer techniques have allowed measurement of volume and size of discrete areas of the brain. Atrophy of the hippocampus (a part of the limbic system) is seen in depression possibly through neurodegeneration. Most antidepressants lead to the production of brain-derived neurotrophic factors that bring about neurogenesis and this could contribute to their effectiveness in depressed patients.
Authored by Dr. Frederic Kalenscher, MD